EXTRACRANIAL CAROTID ATHEROSCLEROSIS IN GENESIS INFARCTION BRAIN IN THE BORDER ZONES SUPRATENTORIAL LOCALIZATION

Vekoslav Mitrović ,
Vekoslav Mitrović

Faculty of Medicine Foča, University of East Sarajevo, Lukavica, Bosnia and Herzegovina

Ramil Marić ,
Ramil Marić

University Hospital Foča, Bosnia and Herzegovina

Snežana Filipović-Danić ,
Snežana Filipović-Danić

Medical faculty, University of Priština - Kosovska Mitrovica, Mitrovica, Kosovo

Novica Petrović
Novica Petrović

Faculty of Medicine Foča, University of East Sarajevo, Lukavica, Bosnia and Herzegovina

Published: 01.01.2019.

Volume 47, Issue 1 (2019)

pp. 15-18;

https://doi.org/10.70949/pramed201901418M

Abstract

The emergence of brain infarction depends to a great extent on the status of collateral blood circulation and is associated with hemodynamic instability. This kind of brain infarction is usually found in border vascular zones. Infarction of borderline fields arises between vascular territories ACA and ACM or between ACM and ACP, deep paraventricular infarcts in internal border zones, wound or triangular cortical infarcts, as well as major infarctions involving the entire irrigated territory of ACM.

Keywords

References

1.
Norrving B. Oxford Textbook of Stroke and Cerebrovascular Disease.
2.
Gavrilescu T, Carlos C. Clinical stroke syndromes: clinical-anatomical correlations. Vol. 7, Cerebrovasc Brain Metab Rev.
3.
Markus HS, King A, Shipley M, Topakian R, Cullinane M, Reihill S, et al. Asymptomatic embolisation for prediction of stroke in the Asymptomatic Carotid Emboli Study (ACES): a prospective observational study. Vol. 9, Lancet Neurol. p. 663–71.
4.
Reinhard M, Gerds TA, Grabiak D, Zimmermann PR, Roth M, Guschlbauer B, et al. Cerebral dysautoregulation and the risk of ischemic events in occlusive carotid artery disease. Vol. 255, J Neurol. p. 1182–9.
5.
Caplan LR. Caplan’s Stroke: A Clinical Approach.
6.
Hiroshi Y, Takashi K, Yoshihiko K. Selective neuronal damage and borderzone infarction in carotid artery occlusive disease: A 11 C-Flumanezil PET study. Vol. 46, J Nucl Med. p. 1973–9.
7.
Momjian-Mayor I, Baron JC. The pathophysiology of watershed infarction in internal carotid artery disease. Vol. 36, Stroke. p. 567–77.
8.
The European Stroke Organization (ESO) Executive Committee and the ESO Writing Committee. In: Guidelines for Management of Ischemic Stroke and Transient Ischemic Attack 2008.
9.
Amarenco P, Bogousslavsky J, Caplan LR, Donnan GA, Hennerici MG. New approach to stroke subtyping: the ASCO (phenotypic) classification of stroke. Vol. 27, Cerebrovasc Dis. p. 502–8.
10.
Damasio H. A computed tomographic guide to the identification of cerebral vascular territories. Vol. 40, Arch Neurol. p. 138–42.
11.
Moore WS. Fundamental Considerations in Cerebrovascular Disease. In: Vascular Surgery. p. 1882–96.
12.
Hogberg D, Kragsterman B, Bjorck M. Carotid artery atherosclerosis among 65-year-old Swedish men: a population-based screening study. Vol. 48, Eur J Vasc Endovasc Surg. p. 5–10.
13.
Rockman CB, Hoang H, Guo Y. The prevalence of carotid artery stenosis varies significantly by race. Vol. 57, J Vasc Surg. p. 327–37.
14.
Rajiv Mangla BK, Almast J, Ekholm SE. Border Zone Infarcts: Pathophysiologic and Imaging Characteristics. Vol. 31, RadioGraphics. p. 1201–14.
15.
Blackwood W, Hallpike JF, Kocen RS, Mair WGP. Atheromatous disease of the carotid arterial system and embolism from the heart in cerebral infarction: a morbid anatomical study. Vol. 92, Brain. p. 897–906.
16.
Moore WS. Surgical significance and management of the ulcerated carotid plaque. p. 199–212.
17.
Eliasziw M, Kennedy J, Hill MD. Early risk of stroke after a transient ischemic attack in patients with internal carotid artery disease. Vol. 170, CMAJ. p. 1105–9.
18.
Derdeyn CP, Khosla A, Videen TO. Severe hemodynamic impairment and border zone-region infarction. Vol. 220, Radiology. p. 195–201.
19.
Seiler A, Jurcoane A, Magerkurth J. T2-weighted imaging within perfusion-restricted tissue in high-grade occlusive carotid disease. Vol. 43, Stroke. p. 1831–6.

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