More articles from Volume 51, Issue 3, 2022
Anatomical and morphological characteristics of the appendix in acute appendicitis
Coronary sinus, microanatomical study
Analysis of the characteristics of traffic trauma
Understanding wedge resection research productivity with visualization: A scientometric attitude
Prevention of micronutrient deficiencies in the elderly
Fructose metabolism: The pathogenic potential of a little molecule
,
Dijana Mirić
,
Bojana Kisić
,
Dragana Pavlović
,
Ilija Dragojević
Sladoje Puhalo
Abstract
References
1.
Ilic I, Ilic M, Kocic S. Dietary Habits and Diabetes Mellitus Prevalence in Men and Women: National Health Survey in Serbia. Foods 2021.
2.
Grujić V, Dragnić N, Radić I, Harhaji S, Šušnjević S. Overweight and Obesity among adults in Serbia: Results from the National Health Survey. Vol. 15, Eating and Weight Disorders - Studies on Anorexia, Bulimia and Obesity. 2010. p. e34–42.
3.
Koliaki C, Dalamaga M, Liatis S. Update on the Obesity Epidemic: After the Sudden Rise, Is the Upward Trajectory Beginning to Flatten? Vol. 12, Current Obesity Reports. p. 514–27.
4.
Saklayen MG. The Global Epidemic of the Metabolic Syndrome. Vol. 20, Current Hypertension Reports. 2018.
5.
Vadiveloo M, Scott M, Quatromoni P, Jacques P, Parekh N. Trends in dietary fat and high-fat food intakes from 1991 to 2008 in the Framingham Heart Study participants. Vol. 111, British Journal of Nutrition. 2014. p. 724–34.
6.
Park JH, Moon JH, Kim HJ, Kong MH, Oh YH. Sedentary Lifestyle: Overview of Updated Evidence of Potential Health Risks. Vol. 41, Korean Journal of Family Medicine. 2020. p. 365–73.
7.
Imamura F, O’Connor L, Ye Z, Mursu J, Hayashino Y, Bhupathiraju SN, et al. Consumption of sugar sweetened beverages, artificially sweetened beverages, and fruit juice and incidence of type 2 diabetes: systematic review, meta-analysis, and estimation of population attributable fraction. BMJ. p. h3576.
8.
Evans RA, Frese M, Romero J, Cunningham JH, Mills KE. Fructose replacement of glucose or sucrose in food or beverages lowers postprandial glucose and insulin without raising triglycerides: a systematic review and meta-analysis. Vol. 106, The American Journal of Clinical Nutrition. 2017. p. 506–18.
9.
Zafar MI, Frese M, Mills KE. Chronic Fructose Substitution for Glucose or Sucrose in Food or Beverages and Metabolic Outcomes: An Updated Systematic Review and Meta-Analysis. Vol. 8, Frontiers in Nutrition.
10.
Bray GA, Popkin BM. Calorie‐sweetened beverages and fructose: what have we learned 10 years later. Vol. 8, Pediatric Obesity. 2013. p. 242–8.
11.
Bocarsly ME, Powell ES, Avena NM, Hoebel BG. High-fructose corn syrup causes characteristics of obesity in rats: Increased body weight, body fat and triglyceride levels. Vol. 97, Pharmacology Biochemistry and Behavior. 2010. p. 101–6.
12.
Kolderup A, Svihus B. Fructose Metabolism and Relation to Atherosclerosis, Type 2 Diabetes, and Obesity. Vol. 2015, Journal of Nutrition and Metabolism. 2015. p. 1–12.
13.
Barclay T, Ginic-Markovic M, Cooper P, Petrovsky N. The chemistry and sources of fructose and their effect on functionality and health implications. Vol. 3. 2012. p. 67.
14.
Andersen MK, Skotte L, Jørsboe E, Polito R, Stæger FF, Aldiss P, et al. Loss of Sucrase-Isomaltase Function Increases Acetate Levels and Improves Metabolic Health in Greenlandic Cohorts. Vol. 162, Gastroenterology. 2022. p. 1171-1182.e3.
15.
Teff KL, Elliott SS, Tschöp M, Kieffer TJ, Rader D, Heiman M, et al. Dietary Fructose Reduces Circulating Insulin and Leptin, Attenuates Postprandial Suppression of Ghrelin, and Increases Triglycerides in Women. Vol. 89, The Journal of Clinical Endocrinology & Metabolism. 2004. p. 2963–72.
16.
Noelting J, DiBaise JK. Mechanisms Of Fructose Absorption. Vol. 6, Clinical and Translational Gastroenterology. 2015. p. e120.
17.
Sun SZ, Empie MW. Fructose metabolism in humans – what isotopic tracer studies tell us. Vol. 9, Nutrition & Metabolism. 2012. p. 89.
18.
Castelló A, Gumá A, Sevilla L, Furriols M, Testar X, Palacín M, et al. Regulation of GLUT5 gene expression in rat intestinal mucosa: regional distribution, circadian rhythm, perinatal development and effect of diabetes. Vol. 309, Biochemical Journal. 1995. p. 271–7.
19.
Muriel P, López-Sánchez P, Ramos-Tovar E. Fructose and the Liver. Vol. 22, International Journal of Molecular Sciences. p. 6969.
20.
Jiang L, Ferraris RP. Developmental reprogramming of rat GLUT-5 requires de novo mRNA and protein synthesis. Vol. 280, American Journal of Physiology-Gastrointestinal and Liver Physiology. 2001. p. G113–20.
21.
Helsley RN, Moreau F, Gupta MK, Radulescu A, DeBosch B, Softic S. Tissue-Specific Fructose Metabolism in Obesity and Diabetes. Vol. 20, Current Diabetes Reports. 2020.
22.
Ferraris RP, Choe J yong, Patel CR. Intestinal Absorption of Fructose. Vol. 38, Annual Review of Nutrition. 2018. p. 41–67.
23.
GIBSON PR, NEWNHAM E, BARRETT JS, SHEPHERD SJ, MUIR JG. Review article: fructose malabsorption and the bigger picture. Vol. 25, Alimentary Pharmacology & Therapeutics. 2007. p. 349–63.
24.
Jang C, Hui S, Lu W, Cowan AJ, Morscher RJ, Lee G, et al. The Small Intestine Converts Dietary Fructose into Glucose and Organic Acids. Vol. 27, Cell Metabolism. 2018. p. 351-361.e3.
25.
Yu S, Li C, Ji G, Zhang L. The Contribution of Dietary Fructose to Non-alcoholic Fatty Liver Disease. Vol. 12, Frontiers in Pharmacology.
26.
Guney C, Bal NB, Akar F. The impact of dietary fructose on gut permeability, microbiota, abdominal adiposity, insulin signaling and reproductive function. Vol. 9, Heliyon. 2023. p. e18896.
27.
Pinnick KE, Hodson L. Challenging metabolic tissues with fructose: tissue‐specific and sex‐specific responses. Vol. 597, The Journal of Physiology. 2019. p. 3527–37.
28.
Geidl-Flueck B, Gerber P. Insights into the Hexose Liver Metabolism—Glucose versus Fructose. Vol. 9, Nutrients. p. 1026.
29.
White JS. Straight talk about high-fructose corn syrup: what it is and what it ain’t. Vol. 88, The American Journal of Clinical Nutrition. 2008. p. 1716S-1721S.
30.
Iizuka K. Recent Progress on Fructose Metabolism—Chrebp, Fructolysis, and Polyol Pathway. Vol. 15, Nutrients. p. 1778.
31.
Adelman RC, Spolter PD, Weinhouse S. Dietary and Hormonal Regulation of Enzymes of Fructose Metabolism in Rat Liver. Vol. 241, Journal of Biological Chemistry. 1966. p. 5467–72.
32.
Merriman TR, Dalbeth N, Johnson RJ. Sugar-sweetened beverages, urate, gout and genetic interaction. Vol. 20. 2014. p. 31–8.
33.
Nakagawa T, Hu H, Zharikov S, Tuttle KR, Short RA, Glushakova O, et al. A causal role for uric acid in fructose-induced metabolic syndrome. Vol. 290, American Journal of Physiology-Renal Physiology. 2006. p. F625–31.
34.
Zhang P, Sun H, Cheng X, Li Y, Zhao Y, Mei W, et al. Dietary intake of fructose increases purine de novo synthesis: A crucial mechanism for hyperuricemia. Vol. 9, Frontiers in Nutrition.
35.
Liu L, Li T, Liao Y, Wang Y, Gao Y, Hu H, et al. Triose Kinase Controls the Lipogenic Potential of Fructose and Dietary Tolerance. Vol. 32, Cell Metabolism. 2020. p. 605-618.e7.
36.
Basaranoglu M. Fructose as a key player in the development of fatty liver disease. Vol. 19, World Journal of Gastroenterology. 2013. p. 1166.
37.
Dekker MJ, Su Q, Baker C, Rutledge AC, Adeli K. Fructose: a highly lipogenic nutrient implicated in insulin resistance, hepatic steatosis, and the metabolic syndrome. Vol. 299, American Journal of Physiology-Endocrinology and Metabolism. 2010. p. E685–94.
38.
Janevski M, Ratnayake S, Siljanovski S, McGlynn MA, Cameron-Smith D, Lewandowski P. Fructose containing sugars modulate mRNA of lipogenic genes ACC and FAS and protein levels of transcription factors ChREBP and SREBP1c with no effect on body weight or liver fat. Vol. 3, Food Funct. p. 141–9.
39.
Stanhope KL, Bremer AA, Medici V, Nakajima K, Ito Y, Nakano T, et al. Consumption of Fructose and High Fructose Corn Syrup Increase Postprandial Triglycerides, LDL-Cholesterol, and Apolipoprotein-B in Young Men and Women. Vol. 96, The Journal of Clinical Endocrinology & Metabolism. 2011. p. E1596–605.
40.
Baena M, Sangüesa G, Dávalos A, Latasa MJ, Sala-Vila A, Sánchez RM, et al. Fructose, but not glucose, impairs insulin signaling in the three major insulin-sensitive tissues. Vol. 6, Scientific Reports.
41.
Roberts AC, Porter KE. Cellular and molecular mechanisms of endothelial dysfunction in diabetes. Vol. 10, Diabetes and Vascular Disease Research. 2013. p. 472–82.
42.
Softic S, Stanhope KL, Boucher J. Fructose and hepatic insulin resistance. 2020. p. 1–15.
43.
Sano H, Nakamura A, Yamane M, Niwa H, Nishimura T, Araki K, et al. The polyol pathway is an evolutionarily conserved system for sensing glucose uptake. Vol. 20, PLOS Biology. p. e3001678.
Citation
Copyright
This work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License.
Article metrics
Google scholar:
See link
The statements, opinions and data contained in the journal are solely those of the individual authors and contributors and not of the publisher and the editor(s). We stay neutral with regard to jurisdictional claims in published maps and institutional affiliations.
