Allergic disease are actual pathologic condition in medicine. From this diseases can get ill people of any age, both sex, and the number of patients with allergic diseases rapidly increase in countries of progressive world. This disease are wide - spread, with tendency of increase, and they can have fatal result in extreme cases. Because of high prevalence of morbidity and complications they have, allergic diseases are health problem. According to World allergic organization, more than 40 % of population in world will be atopic. In our environment this diseases are increasing, because of bad ecological condition from one side, and from the other side of genetic influence, and on the top of ladder are allergic diseases of respiratory system. The mayor key process in atopia is continuous and plentiful production of IgE antibody. 

In clinical hyperkalemia, correlation between plasma K and the ECG is less reliable. A tall, peaked, symmetrical T wave with a narrow base, the so-called "tented" T wave is the earlinest ECG abnormality, usually best seen in leads II, III, V2, V3, and V4. The tented appearance and the narrow base are probably more characteristic of hyperkalemia than is the amplitude of the T wave. A decrease in amplitude of the R wave, appearance of a prominent S wave, widening of the QRS complex, depression of the ST segment evolve as plasma K approaches 8-9m Eq/liter. With hyperkaliemia, depression of intraventricular conduction is characteristically diffuse and results in prolongation of both the initial and terminal parts of the QRS complex. The resulting pattern may resemble RBBB, LBBB, left anterior or posterior divisional block, or a combination of the four.

Chronic pulmonary heart (HPS) stands for presence of right ventricular hypertrophy with or without right ventricular failure, arising as a consequence of chronic bronchial diseases, lung, pulmonary artery, or severe deformation of the thoracic skeleton. Excludes the pulmonary hypertension as a result of heart disease of left cavities, and congenital heart disease. During the evolution of chronic pulmonary heart, distinguished three stages: a) uncomplicated pneumopathology, b) compensated chronic pulmonary heart and c) decompensated chronic pulmonary heart. Lung ventilation is the process of moving air from the external environment to the alveolar and back, and its ultimate aim is alveolar ventilation space. This process takes place through active action - inspirium and passive - expirium, which causes the lungs to the basic position of rest. The disorders of ventilation lung function can be obstructive and restrictive, and there are often mixed forms with predominantly obstructive or predominantly restrictive pulmonary function disorder. These disorders were particularly expressed in patients with chronic decompensated pulmonary heart, what indicates this work. We studied 40 patients and found low values of vital capacity (VC) and forced exspirium volume in the first second (FEV ), with normal values of 1 Tiffeneau index, which speaks to the fact that respondents with decompensated chronic pulmonary heart have mixed ventilation insufficienty, ìainly restricted type heavy degree. 

Epstein-Barrov virus (EBV) is a cause of heterophile antibody positive infectious mononucleosis (IM),which is characterized by fever, sore throat, limfadenopatiom and atypical limfocitozom. IM is mainly a self-limited disease. Complications of the central nervous system, hepatitis (which can be fulminant), (splenomegalia, obstruction of the upper respiratory tract with bacterial can sometimes be deadly.

A characteristic hemodynamic pattern has observed in patients with right ventricular infarction, with frequently accompanies inferior left ventricular infarction or rarely occurs in isolated form. The electrocardiogram may provide the first clue that right ventricular involvement is present in the patient with inferior wall myocardial infarction. Most patients with right ventricular infarction have ST- segment elevation in lead V4R (right precordial lead in V4 position). ST segment elevation of 0,1mV or more in anyone or in combination of leads V4R, V5R, and V6R in patients with the clinical picture of acute myocardial infarction (MI) is highly sensitive and specific for the diagnosis of right ventricular MI.

Cardiac dysrrhythmias can be found in patient with no signs of ischemic cardiac disease but supraventricular and ventricular arrhythmias can be developed in cardiac hypertrophy. In this work we have examined frequency of cardiac dysrrhythmias in patients with hypertension and with or without hypertrophy of left ventricule (LVH). Investigation was accomplished on 80 patients with high blood pressure. In all 80 patients, basic on ECHO signs we define LVH, and by using of Holter monitoring we analised dysrrhythmias. Basic on ECHO signs we have 2 groups of patients: I.group ECHO (+) LV, and II group ECHO (-) LVH, in each 40 patients. Holter monitoring have 37 patients [22 mans (59,45%) and 15 women (40,54%)]. Hypertensive patients with LVH, who hade dysrrhythmia, have middle value IMLV 155,3 ± 27,21; аgе 56,42 ± 6,04; duration of hypertension 11,73 ±10,05; and EF% 55,2 ± 3,77. In patients with Holter monitoring 16 ( 43,32%) have dysrrhythmias type II, IIIa, IIIb and IV Lowny degree, (12) 34,43% from 43,32% have LVH. Salves VES are registrated only in group hypertension patients with ECHO(+) 5,4%. By using Mann Whitney statistic test for presents of dysrrhythmias in 2 groups of patients (ECHO LVH and ECHO without LVH), we found statistics significants (p = 0,049). In IMLV 60-110 g/m² 8,82% patients have dysrrhythmia, IMLV 151-200 g/m² dysrrhythmias are registrated in 40%; and 50% patients have dysrrhythmias in IMLV> 200g/m². In group hypertensive patients with LVH dysrrhythmias are registrated in group with ecscentric LVH 35,1%

Chronic pulmonary heart (CPH) stands for presence of hypertrophy and/or dilatation of right heart ventricle caused by pulmonary hypertension as a consequence of diseases that are damaging lungs function or structure. By chronic pulmonary heart are not considered those hypertrophies of right ventricle which are consequences of left cardiopaths or congenital heart anomalies with skips. During the CPH evolution three stages were formed: a) uncomplicated pneumopathology, b) compensated chronic pulmonary heart and c) decompensated chronic pulmonary heart. The disorders of cardiac rhythm are often at decompensated CPH, which is proved by this scientific work as well. 40 sick people diseased of decompensated CPH are treated in purpose to establish the presence and the characteristics of cardiac arrhythmia at these patients. It is established that the major percentage of frequency is made by sinus tachycardia, atrial extrasystols, and ventricular extrasystols. Of 40 examinees 65% had sinus tachycardia, 37,5% atrial extrasystols, 30% ventricular extrasystols and 20% combined atrial and ventricular extrasystols. This proves us that there is a significant percentage of cardiac rhythm disorder frequency at the sick people of decompensated chronic pulmonary heart.